Welcome to the youtube channel from Dr. Paul Merritt, a research psychologist specializing in memory as well as drugs and human behavior. I am in the process of sharing lectures I have recorded for my on-line "Drugs and Human Behavior Course." I hope you will find them informative and encourage you to share with any friends or colleagues interested in learning more about how drugs work and how they affect both the brain and our behavior.
I nearly died last night in hospital from not a huge amount of speed. I've done it for years and never had a bad reaction. Now I've damaged my heart and they said I very nearly had a heart attack. They said if I had any more I could've very possibly died. I'm 21. Please be careful guys you don't realise how easy it is until it happens to you.
Great talk but: Please, please consider re-recording when you don’t have a cough. It’s phenomenally distracting. Maybe you could edit out the coughing and repost
The opponent process theory is wrong. You say that the afterimage of red is green, and for green it's red. But that's not true. The afterimage of red is cyan, and for green it's magenta. This proves this and some other assumptions of the opponent process theory wrong.
You mentioned that In regards to Suboxone the buprenorphine is responsible for controlling the withdrawal symptom, while the naloxone is responsible for suboxone's blocking effects. This is not accurate, the buprenorphine is responsible for all of the effects in Suboxone, and the naloxone is basically inert. The reason for this is because buprenorphine has a far greater binding affinity at the opioid receptors compared to naloxone. Because of this none of the naloxone is able to reach the receptors as they are already saturated by the buprenorphine. So it is buprenorphine and buprenorphine alone that is responsible for the blocking effects of suboxone and why Subutex and other mono formula versions are still effective. It's also a big misconception that the naloxone contained in the Suboxone will throw users into precipitated withdrawal or have blocking effects if they were to try and abuse suboxone via injecting or insufflation. This too is inaccurate and for the exact same reason, buprenorphine simply has far too high of a binding affinity for naloxone to push past it especially in the dose ratios contained inside Suboxone. In 99% of cases the effects of Suboxone and subutex are exactly the same no matter what the route of administration is. These misconceptions were started when Suboxone was released as they used the naloxone and these "myths" regarding it to make their product stand out from the rest helping it get faster approval from the FDA. While at the same time utilizing it as a marketing strategy to both medical providers and patients, setting suboxone aside from the rest of the bupe medications this boosting their sales. In reality the naloxone provides no benefits whatsoever to the medication. It is important for this misconception to be clarified especially amongst medical professionals because there are times that Subutex can be a much better form of treatment. For example naloxone Even when it is not active at the opioid receptors can still cause negative side effects to some people who are allergic or don't tolerate it well, like a migraines for example that is one very common side effect of Suboxone and it is due to the naloxone it contains. Also Subutex tends to be a much cheaper alternative which is beneficial for many people suffering from OUD. Yet many doctors are unwilling to prescribe it due to this very common misconception started by the creators of Suboxone. this is one of many misconceptions surrounding addiction in general, and one that definitely needs to be clarified and corrected because many people are negatively impacted due to the ignorance of their providers.
I'm "going" twice 😁 bc I'm listening further, as I write and I WANT to again say THANK YOU IMMENSELY for this!!! What your explaining this second half is exactly the kind of information I was looking for but doubted I'd get. I thought it'd be too difficult to put into words since it's abstract. AWESOME and PRICELESS information here!!!
Yes, very interesting and PUZZLING indeed. Pretty difficult to fully understand from their perspective. My poor kitty is actually experiencing this after a stroke. So I thank you VERY much for sharing your education with us.... especially since it's not at all straightforward in understanding. This is great in helping us understand their world a bit better so we can hopefully assist them better.
Some of this is bogus, nobody puts mushrooms on their pizza, bevause the ovens heat will degrade the sensitive tryptamine molecules. And it's also not a lipophilic compound it's soluble in water and ethanol. More stable in ethanol solution though. In fact there is a procedure called "lemon-tek" where people soak the mushrooms in a tiny bit of lemon juice, to acidify and extract the psilocybin and psilocin as well as partially convert the psilocybin to psilocin, and causes a faster onset and overall more compressed pharmacokinetics. Cooking with lipids is for THC! I don't mean to barge in and start being professor over here, but this is my niche, and my BS meter went crazy. I don't blame you tho just your source. My sister got a minor in addiction and recovery studies at A&M and their curriculum had a lot of straight up misinformation that seemed straight out of the Reagan Era. I study pharmacology, so we often got into fights about things like thisb 😂
Your information about the potency of cannabis is extremely outdated. 99% of the available cannabis that isn't brick weed is gonna be at LEAST 10% at MINIMUM and often 12-15% or even 20%. Some strains can be as high as 25% THC. In this day and age (and even 4 years ago) it would be a challenge to find cannabis that low potency. Also Your slide said indica has more thc than sativa but this is not true. They can both have wildly different contents.
@13:45-14:00 you put Antabuse in the substitute category. Antabuse is not an alcohol substitute nor is it even psychoactive. It inhibits Acetaldehyde dehydrogenase, causing a buildup of Acetaldehyde which is extremely unpleasant, and is supposed to cause aversion to alcohol. Can be dangerous though. Either way it's not a substitute! Acamprosate however is somewhat closer to a substitute in that it has effect on GABA and NMDA like alcohol and therefore works with the same receptors.
I’m studying psychology, and our course is biological psychology, it’s so hard to understand the concept of topics as my first language isn’t English, but this is much more better than what professor explains in class 😅 thank you so much for your great work !
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I find it very interesting current theory indicates culture affects memory. It seems our government currently wants us to ignore culture and focus on skin color. That is, comparing cultures is taboo. In this presentation I heard nothing about the effect of emotion on memory -- why recalling some memories can put us back in the event. Also, no mention of the associative nature of memory -- why we recall what we do and when we do -- what triggers memories. Or, why we have trouble at time recalling memories.
I knew a guy that huffed Freon and said it was crazy and after a while he was in the hospital and they told him he almost died etc. but out of all the things that could have killed him huffing Freon he almost died from hypothermia of all things then I looked it up and apparently, yeah that happens sometimes when you huff Freon
This lecture is great, thank you. I know I’m late, but I was wondering if there was a bibliography to consult regarding this lecture? Or perhaps some suggested books? Thanks in advance
Hi, just wanted to say thank you so much for your lectures. I'm studying to become a psychology researcher. I am a special needs learner and my current lecturer is difficult for me to understand, but I understand your methodical and organized approach to overviewing this subject much easier for me to understand. Plus, it's helpful to have subtitles and hear a voice in conjunction.
