I just needed this thing. I decided to look the nephrology part of my internal diseases topics for the exam and I was a bit confused with the glomerular disorders,now it's all clear. Thank you for this amazing video! :)
one thing that helps me is the physical location of things on the white board and where you write them. I don’t mind the new animation, but is it possible to keep the same spatial layout with your new animations?
Great point! This is not our new way of presenting our content. Just a future sneak peek of how we will present our lectures for exam preparation! We will continue to use the whiteboard for all of our content as we move forward through clinical medicine.
Hey sir Please make videos about teeth anatomy and histology We really need them Videos r very rare about our study 😢 Ur videos always make me love study man i hope u make videos about these topics
Thanks very much for these videos (SEE FEEDBACK BELOW) as always. Although I too like the old "white board" teeaching method better, I do understood for production efficiency and for saving lectures digitally the Power Point type lectures are better ; HOWEVER, I have a suggestion. If possible, as it seems you have room on most slides (especially on "left side" column points) and disagrams, PLEASE *INCREASE THE FONT SIZE** OF DIAGRAM LABELS, SUB-POINTS AND CHART COMMENTS..... THANKS
I've never understood why nephrotic syndrome leads to hypercoagulation and not haemorrhagias I mean... besides anticoagulant proteins you also urinate coagulation cascade proteins that not only are self-limited by proteinuria and so less synthetized but also the fact it requires just one of the proteins to be missing to elongate aPTT/PT
Here’s the thing- In nephrotic syndrome, the kidneys lose both clotting and anticoagulant proteins in the urine, but more anticoagulants like antithrombin III are lost because they’re smaller and easier to filter through damaged kidneys. To compensate, the liver makes more clotting factors, creating an imbalance that makes the blood more likely to clot, leading to a higher risk of thrombosis instead of bleeding:)
@@Dr.NigelLeonMartis Thanks for your reply, I see the imbalance in concentrations of pro/anti coagulant proteins but I don't see direct link between lost AT III and synthesised factors. I guess liver favours synthesis of coagulation cascade factors in spite of lost AT III. Is liver more sensible to their blood level? 🤔
I developed hypophosphatemia. A 24hr urine collection shows elevated phosphate levels. Kidney function is normal and all other blood tests have been normal. Any thoughts???
