How To Read A Brain MRI - Neuroradiology Made Easy (Maybe?) 

YW. Glad it's helpful.

I'm sure the mnemonic is pure coincidence. 😉 Glad it's helpful.

Thanks!

Anytime

Thanks. Will do.

​@@neuroradish...sir...can signs of watershed stroke be missmatched for something else

And what?

YW. I'm glad it's helpful.

@黃紹閔 Hi, sorry didn't catch your question earlier. If a cystic lesion is similar in signal to CSF (bright on T2 and dark on FLAIR), then yes, it has to do with its T2 property in that TR/TE is same or very similar to water, allowing it to have FLAIR signal suppression. It is not due to fast flow. In theory, if you have a FLAIR hyperintense fluid structure that is flowing rapidly (can't really think of a good example in pathology), then you can have loss of sign ("flow void) too and will appears dark on FLAIR sequence.

If you're referring to a normal MRI (17:14), that slightly brighter T1 signal in GP compared to the adjacent putamen is due to myelin. GP is more myelinated compared to relatively unmyelinated structure like putamen. Which is also why it looks darker on T2 (27:49), as well as GP looks more "pale" on gross anatomy specimen compare to putamen, I believe. If it's a lot brighter, then it is probably pathologic, or due to increased calcification / mineralization deposition.

@@neuroradish Thank you for responding! It was a normal T1-weighted MRI without contrast...I hope I'm saying that right haha. So if GP is already myelinated but the MRI found that it is a "higher t1 signal" than normal range or whatever. Does that mean MORE myelination? The reason I ask is because my neurologist had ordered the MRI due to her suspecting MS. But that's DE-myelination as far as I understand. So that would be like the opposite, it sounds like.

I'm pretty sure it was just a coincidence, but they're definitely bad diseases. You know, bad stuff.