A review of hypocalcemia, including its clinical manifestations, etiologies, diagnostic approach, and treatment. Major topics include vitamin d deficiency, hypoparathyroidism, and secondary hyperparathyroidism.
thank you for another great lecture... I recently had a patient with Severe Acute Pancreatitis with Hypercalcemia as the most attributable cause... scanning through her files, we found that her GP had prescribed her Vit D sachets of 60k units weekly for 8 weeks... that too empirically...
I've been having cramps and numbness in my hands feet and face for a few years. when it's bad my hands will cramp closed and the rest of my body will tighten and lock. I was told it was dehydration/potassium/ magnesium. I did the cheek tap test and my lip twitches...could it be calcium?
Thank you , good lecture. I ran into your clip said May be no need to correct Calcuim ion , I didn’t read the paper look too difficult to me :) , so you mean just take the lab result as it it ?
I bought a Calcium supplement last night and it is Tricalcium Phosphate. Is this okay to use? I'm not calcium deficient, it's just on the low side of normal (54 year old woman) with results at 9.1. Thank you!
While I can't give any specific advice regarding a viewer's specific medical issues, I can say that most physicians regard tricalcium phosphate as equally effective and with no additional significant precautions as compared to the more commonly used calcium carbonate or calcium citrate. As always, any med or supplement should be discussed between a patient and his/her physician/NP.
Please help me, i will report this on our class, and our reference should be Harrisons internal medicine book, but I can’t understand whatever is written on that book
Calcium is a positively charged ion that contributes to the resting membrane potential. Without it the inside of the cell becomes more positive (relatively to the outside) resulting in a state that has an easier time reaching threshold
in resting membrane potential sodium enter inside the cell in small down its concentration gradient through sodium leaking channel ,the calcium guarding this channel prevent entering of sodium. in case of hypocalcaemia more positive charge sodium will enter the cell making inside of the cell less negative so reducing RMP and the value become closer toward threshold( more excitable) ..so the increase excitability in NMJ resulting in involuntary contraction of some muscle (tetany)
Any sufficiently significant electrolyte abnormality can impact fertility as it could impact your entire body's physiology, but I am not aware of any specific associated with hypocalcemia per se. (Nor did a quick search of the medical literature turn up anything.) This is just my speculation, but I doubt that subclincial hypocalcemia (i.e. hypocalcemia mild enough to not cause symptoms) has any measurable impact on fertility.
