Thanks for this video, very informative. However, the issue I was interested in, i.e. cptsd/developmental trauma was not really addressed. Andrew began to respond, around minute 19:55, saying that the cptsd brain doesn't produce EEG cell responses, which I understood are important for neurofeedback training, but he didn't actually finish the response, as he went off on a tangent and then you asked him another question. So what happens when the parts of the brain that produce the hypervigilance don't show up in the EEG? How does he work with that? I would really like to know.
Thanks for the follow up question! Short answer - you *can* see some of the areas that are "irritated" by cPTSD, and classic anxiety areas may still be involved. The amydala and a fwe other sources like the periacqueductal gray do not make EEG, but they have nearby tissue that does, and it is connected and involved in regulation. ie.. you might be training the peri-amygdalar tissue instead of the actual amygdala, but the training on that system is stil effective.
How does this relate to Cognitive decline concerns? I work with many dementia patients, their families and devastated care givers, as an RN Care Coordinator and notice that providers are under the impression that there just are not any options or support for Neuro-psychiatric evaluations, diagnosis and treatment options. Please tell me they are wrong!
There are some things you can do! Once showing as really progressed dementia, things are much harder to change, but up until that point the Apollo Recode program and other metabolic biohacking strategies may make a big difference, plus there are tools like photobiomodulaation and nootropics that may have some impact, and growing lists of pharma interventions that seem to be having some impacts. A QEEG brain map can also help you watch things like speed of processing and fatigue, and distinguish that from "true" memory + aging issues.
