Finally! Here is the long awaited pulmonary/respiratory review for the USMLE (primarily for step 2)!! With over 100 high yield questions, you will be prepared on exam day! Slides (for now) at: ajmonicspdf.co...
Thanks for the video, I am preparing for step3. Your videos are an awesome tool for a quick review. Thanks for the good work. Just sharing my thought for question 101 Increase mitral flow velocity is in mitral regurge, not stenosis. Exertion dyspnea could be due to left ventricular failure. Mitral regurge is the most common valvular lesion in rheumatc heart disease. Thanks 😊
Hello! I would appreciate some clarification on question 79 at 38:59. How can you tell it's bronchitis rather than postnasal drip? I understand that postnasal drip typically presents with upper respiratory symptoms, while bronchitis involves wheezing and lower respiratory symptoms. But none was mentioned... Thank you!
Yeah I am confused about this too. I think its timeline. Bronchitis occurs within a week or two of URI or never resolves, while UACS is more like weeks-month after having URI
I was thinking more so post nasal drip which can also manifest at night as a cough and common after URI. I actually remember a question similar to that one on Uworld and the answer being 2nd gen antihistamine, so I think he might have made a mistake.
@@AJmonics OOh WOW, I'm really glad to hear about Step-3. Can I plz request the first vidoe to be made on CCS cases?, and when can we expect the start of Step-3 plz😊
7:42 there were also serious treatment related events of about the same magnitude, so to me it's a bit of a wash. You can bump up the immune system with nutrition to protect better against severe rsv but you can't necessarily prevent side effects of the antibody shot. 15:43 50mmHg is mild/moderate hypercapnia, to suggest intubation sounds ridiculous (given how severe of a procedure that is) I'm reading asthma guidelines and it sounds like fear mongering. When I am sedentary for a while I frequently feel the need to breathe rapidly (I would say easily 30 per minute), which goes away after working out a little bit. Coincidentally, exercise significantly raises cortisol and epinephrine, exactly the same as the two medications to treat asthma: corticosteroids and beta receptor agonists which nor/epinephrine targets. Exercise also raises tissue and brain co2, which might also relieve high breathing rate. According to guidelines I would already have a 'acute severe asthma attack' at 25 breaths a minute, because I used to have asthma as a child. Asthma deaths at US schools is less than 2 per year from 1990 to 2003. I mean it can't get any rarer. Looking at all the asthma medications, which all simulate exercise induced changes, you'd think the treatment for asthma is exercise (and cause, lack thereof) 22:48 another ridiculous criteria: if a patient has nocturnal waking on any night, then they have persistent asthma? You can wake for a million reasons not related to asthma.
