During that time, it's still immediately after diastole and the pressure in the aorta is still the diastolic pressure of 80 mmHg. After the valve opens and the blood in the ventricles are pushed out, that is when pressure rises upto 120 mmHg as seen in the graph. So when the aortic valve opens, its still 80 mmHg in the aorta.
Please answer this question. I think all textbooks show it wrong. SV increases as EDV increases and they all shorten to the same ES point on the ESPVR curve. Why should it shorten to the same end systolic pressure or point? If you increase SL (as in higher EDV) you should get more force (Force-length relationship) and more force means higher pressure. If they all shortened to the same ESP when you increase EDV, how would they even get the ESPVR curve?
Thank you so much ,with all the illustrations it literally made it so easy to understand,great video 👍🏼👍🏼 Refreshed my medical school knowledge in no time 👍🏼
I see you posted this a year ago so I'm late to the game. A is where the mitral valve opens because the pressure in the ventricle is lower than the pressure in the atria. The segment from A-B, where pressure continues to drop even though the ventricle is filling occurs because the ventricular muscle is continuing to relax. From B to C, you get more filling and only a modest increase in pressure (Pressure higher than point B) because even though the ventricle isn't actively relaxing, in a healthy ventricle its still highly compliant. Then ventricular contraction starts at C resulting in closure of the mitral valve and the initiation of isovolumetric contraction.
