I don't think you understand how much I needed this, today especially. I'm prepping for my pharmacology exam this week and this helps solidify everything in my mind. Thank you so much Medicosis! and thank you Maria :D
What an introduction.. I wasn't in class for this lecture..but damn I bet my professor won't start this way. You are the GOAT at this Job .. period!!!!
In a sepsis you have distributive shock . wow It is a well done explanation. if I want to read more about sympathetic and parasympathetic system, what book would you recommend? or perhaps a paper? Greetings from Peru by the way
I just remembered, when i was a kid, i used to get injured and one time i got hit in the head which caused bleeding and back then old folks whould manage that by compressing the wound site with guess what, wet coffee ground 😳 And i can't help it by noticing thanks to this video that caffeine actually acts as a vasodilator (so not of a significant way of stopping bleeding) but i still wonder where did they got the idea from and can it be, as opposed what i just mentioned, beneficial in some other way in treating wound bleed? Nevertheless, it keeps boggling my mind how drugs can act differently based on their dosage and route of administration (nothing in this life is straightforward, NOTHING!)
1:00:31 if the venous return drops, while contractility increases, can we be sure CO increases? without a good preload, isnt that CO's gonna drop in accordance?
As per my knowledge, Higher Contraction gives more output . As For normal it's about 70ml Remaining in Ventricle after Ventricular Contraction , but if Contraction increases this value of Remaining 70 ml will be less in Ventricle and CO will be More. So in This Venous return is less but the Higher contraction will compensate to give higher CO
