Here we go beyond lifeless diagrams and make medical concepts come alive with animations. Things are much easy to understand when you see them happening right in front of you.
Hi, I am Dr Vipul Navadiya, and I am uploading animation videos for medical education. The target is to translate entire medical literature into animations. Join me in this marathon.
You can also follow me at: • Facebook: facebook.com/NonstopNeuron • Instagram: instagram.com/NonstopNeuron/ • Twitter: twitter.com/NonstopNeuron
Dr Vipul Navadiya, Assistant Professor, Department of Pharmacology, Kiran Medical College, Surat, Gujarat, India.
It was difficult for me .reading and consuption topics guytum and hall now you help me and make All eazer now i can read eazer and All concepts i consume with smart ....i cant say you one time thank and must say thanks handerd and million times ❤❤❤❤❤❤❤❤❤❤
i love your original voicing so much🥺 this voice makes it sound so generic idk i just lose focus😂😂 nevertheless amazing videos, in my opinion the best physiology explanations especially with animations like its even the best animations. also i love summaries in the end of a video, best way to solidify the information or make sure i understood the topic. keep going man i wish u all the success💗💗💗
2:12 Wait, schwann cells and oligodendrocytes are not same. Schwann cells are suppose to be present in peripheral nervous system neurons and oligodendrocytes in central nervous system neurons. They perform same function of myelin formation but schwann cells are also perform phagocytosis and repair. Please correct me if I a wrong.
Sir , as in cholinergic system M3 receptor is for cilliary muscle contraction - near vision, Which adrenergic receptor is responsible for cilliary muscle relaxation,which is need for far vision ,..?
The amount of calcium ion released during each contraction is not completely pumped back into the sarcoplasmic reticulum as the second stimulus is applied within 10 s. Therefore, some residual amount of calcium is left prior to the next stimulus. This increases the quantity of calcium available to the contractile machinery in the next stimulus (the calcium left in the previous contraction plus the calcium released by the present stimulus). Therefore, the second stimulus evokes a bigger response. Similarly, subsequent 2 to 3 stimuli progressively increase the magnitude of contraction. 2. During the first contraction, the temperature in the myocardial cells increases which enhances the enzymatic activity for the muscle contraction. Therefore, the second stimulus acts under the beneficial effect of increased enzymatic activity of the first one. Similarly, few subsequent stimuli progressively increase the height of contraction. 3. The first contraction decreases the viscosity of the sarcoplasm due to increased temperature. Thus, the resistance provided by the sarcoplasm for the second and subsequent contractions is significantly lower than the first one. This increases the magnitude of few subsequent contractions
my videos are slightly more optimized for repeated watching. So they are slighly fast paced. But when watching for the first time, you can decrease the speed by RU-vid setting if you wish.
Kindly re-check and confirm, the passage of molecules through channels/pores is a facilitated diffusion not a simple diffusion as mentioned in the video.
If the Na+/K+/2Cl- cotransporter is being inhibited, doesn't that also leave more potassium in the thick ascending loop of henle? That almost makes more sense to me than more sodium in the collecting duct leading to more being absorbed in the principle cell and leaving the collecting duct more negative thus more potassium excretion. If more sodium is being absorbed from the collecting duct into the principle cell, wouldn't water just follow and negate the action of the diuretic?? The explanation with the principle cell seems more applicable to aldosterone antagonists.
First you need to understand that these processes are NOT "all or none". It's about fine tuning between two extremes. A slight change in the reabsorption pattern is multiplied over time. Talking about your specific confusions: 1. Potassium reabsorbed by Na-K-2Cl cotransporter, is secreted into the lumen normally also. 2. Not all the remaining extra Na is reabsorbed at the collecting duct. So the effect of diuretic is not nullified. I hope it helps.
@@NonstopNeuron makes sense! I guess it would also make sense that any of the sodium leaving in the collecting duct would be replaced by potassium to retain electroneutrality in the lumen.
That makes so much sense. I memorized many different interpretations of EKG abnormalities, but I always felt like I didn't really grasp the basics, even after watching tons of videos and reading a lot. You explained it so clearly, literally made me cry for joy. Thank you so much!
I wonder why you don't mention the activity of any voltage gated potassium channels along the axon and at the nodes of Ranvier. Is it in order to keep the illustration simple that those channels have been omitted? Thank you!
