Lipid Transport (pt. 2) 

With B48 it enters the Blood stream With C2 it acts on the Capillary endothelium of muscle &fats With E it Enters into the liver

I remember Apo B48 = Chlymicrons as, "I just ate, so I have B48" (ate = 8) bc Chylomicrons are only present following dietary Triglyceride consumption.

Bro I can't believe you are providing such quality content for free! You are a life saver and all the future physicians are in your debt. God bless you man.

i dont believe that 3 people disliked this ig they are the teacher who explains everyting in a complex fucking way

I'll forsure make sure to tell all my friends at my med school about your videos great videos man

Dr . D , God bless you 👍. Thanks for the great videos,

Seriously thank you! You’re a godsend

Thank you for this beautiful video! You made it really easy to understand and remember.

What are you man where do you get all these incredible mnemonics this is unreal

its great dude , love the way you explain

Omg, this was a fantastic way to break it down. And the mnemonics for lipid transports are way too good. I've written all of them down on my First Aid.

After multiple attempts to learn, I’m beginning to have a grasp of it finally😊thanks a thousand times

Phenomenal as always, thank you dirty

Sir, please make a video on glyco-lipid metabolism. Your videos are so awesome!

All concepts are clear. Thank you!!

omg dirty medicine how are you so good at this..

Thank you for the great videos, :)

hi dirty thank you for all the videos. I wonder if it is possible to integrate this video to lipid lowering drugs? because drugs are also highly tested. could you make a video about lipid lowering drugs as well? thank you so much!

B48 = transports into lymphatic system, HDL adds C2 and E, in the blood circulation. C2 activates LPL to release the triglycerides for storage E then then break them into remnants for recycling. The chylomicron goes into the liver where it will be recycled.

Love this explanation

What an explanation, great work🙀

I dont get why insulin cause breakdown of triglycerides to form fatty acids i though this happen by glucagon or something during starvation... activating lipase shouldnt insulin promote triglycerides formation due to excess glucose in blood

These are some top videos bro 👍🏻

thanks man I can’t thank you enough

Thank you.

Very very very helpful !!! 👍👍

those 4 people disliked this because they thought the thumbs-down button is download

You are my friend dirty medicine that for making me understand this difficult subject on lipid transport. I love uuu

this is SO GOOD

This is really helpful, thanks☺

Hi Doctor Are there any videos about Malate Aspartate shuttle or glycerol phosphate shuttle and do these pathways come in USMLE or never?

Mate. Did the HDL Arrive on a magic carpet?

GOAT stuff 🙌🙌

Nice video. Tks a lot

im starting to believe that the bomber is the exact reason why they named apob48 like that :0

Very nicely described

Cool .. thanks ..

thx

doesn't Insulin 'drive' the fatty acids into the cells rather than 'break down' triglycerides into fatty acids? and I think ApoE is the key that allows the remnant Chylo. to "Enter" into the liver.

Weldone

Hello, can I get some screenshots of your illustrations for my presentation in my class? The illustrations you made are so easily understandable. You made it very clear thank you. Of course, I'll put the link to your channel for the source

u have simpliefied this so much. am watching this while reading fa and its such a cluster fuck. THANK YOU SIRRRRR.

❤️❤️

Chylomicrons? What is that? Chylo=juice micro=micro?

What reference do you use?

Please make more videos n

Good vids, though a little bit too much of repetitions. (I'd rather like to get the whole picture quick, then potentially re-watch it)

I love you

Can we please have part 3 lipid transport sir I am in dire need of that exam soon

8:25 threw me in for a loop man, wasted like 20 minutes trying to understand your whole explanation cause your're not wrong but you're also not entirely right either lol let me explain: You say that in type 1 DM you are unable to store fats because insulin mediates the breakdown of fatty acids and storage of said fatty acids which technically is like 10% of whats really going on. The true cause of the weight loss is the unnopposed action of glucagon which activates HSL within the adipocytes, and this in turn metabolizes stored fatty acids for energy production since these patients are unable to yield energy from the glucose . Like i said you're not wrong your wording is just confusing. Great video though honestly, I wouldnt have made it through these first 2 years of med school without some dirty little medicine, love your content my guy!

This shit sucks, thanks