Lp(a): Assessing and Treating a Major Source of Atherosclerosis - CME Recording

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Dr. Trippi and Dr. Brown give a short introduction of the current understanding of Lp(a) patho-physiology, a discussion of treatment options at present, a thorough discussion of how lipoprotein apheresis fits into the treatment landscape, two case histories of patients receiving lipoprotein apheresis for increased Lp(a) and a short discussion of medications in development for the treatment of Lp(a).

Опубликовано:

15 фев 2024

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Комментарии : 14

@beepbeepnj2658 4 месяца назад

Lp(a) is in your genetics to live a long healthy life. "Remarkably, one-quarter of the centenarians had high Lp(a) serum levels even though they never suffered from atherosclerosis-related diseases." 1998 G. Baggio. Low levels of Lipoprotein(a) are not good. 2012 article title: Low Lipoprotein(a) Concentration Is Associated with Cancer and All-Cause Deaths: A Population-Based Cohort Study (The JMS Cohort Study) The Lp(a) levels were inversely correlated with the CIMT in this population, suggesting that subjects with a low Lp(a) level may have a predisposition to carotid atherosclerosis. This finding was preliminary and should be investigated further in larger studies and in additional settings. 2012 article title, CIMT thickness in asymptomatic subjects with low Lipoprotein(a) levels.

@user-wf2ho3kd9n 4 месяца назад

I am 79 and no event but LPa 217 and never knew about it until 2 years ago but have been put on 5mgs Crestor and 10 mgs Zetia plus a 81 mgs aspirin daily. I feel fine and fast 18 hours a day and eat low carb/ Mediterranean. I am 5’5” and 128. All blood tests are great except LDL was 152 but now 80. APOB 80 and HDL 116 and Triglycerides 63 all prior to starting meds.

@HuyLe-up3mx 4 месяца назад

@@user-wf2ho3kd9n Thanks for sharing Goes to show that lifestyle is very important

@davidmusial1611 4 месяца назад

The chart doesn't say how much niacin was used. This is typical md bs.

@monnoo8221 4 месяца назад

Lp(a) is NOT a cause of athersclerosis. It is best a mediator, s it prevents clots from being dissolved. point i, one has to avoid injuries of endothelium in the first place. Prevent high glucose, prevent inflammation, eat low carb to elevate HDL = preventing the breakdown through triglycerides, keep hmocystein in check via folate B12 and betaine.... Secondly, ordinary vit C is strongly inversely correlated with Lp(a). you are wasting 1h+ of time

@JMK-vo8pv 4 месяца назад

You nailed it, monnoo! There is ABSOLUTELY ZERO PROOF in any study, any research, any literature that Lp(a) particles actually CAUSE atherosclerosis. The only thing that the Lp(a) zealots have to keep pushing their bogus theory on Lp(a) is a bunch of ASSOCIATIONAL data that proves nothing. As the well respected cardiologist, Dr. Nadir Ali says, "Lp(a), just like LDL, is a 'firefighter' and has nothing to do with CAUSING atherosclerosis/plaque". Dr. Nadir Ali reports that he has performed coronary angiograms on some elderly people with lifelong elevated Lp(a) levels and these senior citizens have perfectly clean coronary arteries!