Hey Guys, Definitely want to thank you very much for these videos. Been constantly getting 95% on my university exams. keep up the good work. Time for finals!
This was VERY helpful! I'm in nursing school and your videos/drawings/explanations really help me grasp the material. I really liked the RAAS and Adrenergic Receptor videos too. Please continue making videos; thank you!!
Thank you for an excellent explanation. Which impact has a low potassium level on all this, f. ex. the heart? Thank you for an answer. (Or have you already made a video on that subject?) Modern diet: high levels of sodium, low levels of potassium.
Amazing !!!! Why yout weren't my professor. I left Zoology just because of hopeless teachers who were getting confused while teaching us and making us confused 😕
Hi i have a question i hope you will answer if the NA goes inside the cell for the CA goes out of the cell then how does the CA goes back again in exchange of what? And if NA goes into the cell in exchange of CA how does the potassium goes outside again in exchange of what? Hoping you will answer my question doc Thank you
Hello i have question if u can help plz KCl concentration in a solution that surrounds an isolated cell was increased. How will resting membrane potential (RMP) and cell excitability change in this case? A. RMP decreases, excitability increases B. RMP increases, excitability increases C. RMP increases, excitability decreases D. RMP decreases, excitability remains unchanged E. RMP and excitability remain unchanged
I don't quite understand something about cardiac glycosides. For example, digoxin inhibits the Na+/K+ ATPase pump which leads to a buildup of Na+ in the cell. When a cell has a lot of Na+, it activates the Na+/Ca2+ pump where it exchanges 3 Na+ ions for 1 Ca2+ ion. How can digoxin be lethal if this is the case? There's still calcium flowing into the cell!
It would be ideal if most if not all GPs...specialists and alike possessed this depth of knowledge..but sadly though its not the case. So many so called " doctors " forget what they learnt at medical school and when questioned about how a drug actually works..or how a body system carries out its task they fail miserably. Remember at uni all medical students have to only pass their subjects with a mark of 55 throughout their course to graduate..that means some come out not knowing 45% of their material...which doesnt take a genius to work out how dangerous that could be to us..their patients!!! The sad and laughable irony of it all is that to get into medical school you need to practically have almost perfect scores in their HSCs and a umat or gamsat score equally as high. Crazy but true! At uni i kicked the arses of nearly all the medical students in pharmacology which was my specialty back then 30 years ago. Just wished that doctors ( actually dont like calling them that because its an honorary title..unless they actually do have a doctorate in medicine) ... the new courses in oz are now reflecting that dilemma..which is good i suppose..but its mad that we call..dentists..vets..and even optometrists drs....people incorrectly assume that all doctors are healers of people when in fact its not the case...anyway i salute Dr Mike who is an example of what it truly means to a doctor!!!
But, but, but, but, but......even though the Na/Ka ATPase pump is so important, you are not explaining how we can improve/lessen it's effect. In other words, every atom in our body is important, going along with your logic! Yeah, we know that. :) Can you enlighten us as to how we can apply or alter the Na/Ka ATPase pump? Diet, exercise, etc. (Note, several doctors (neurologists) have told me diet cannot alter this Na/Ka ATPase pump). I am not sure, but not a doctor.
