What is Cancer? 

1:37 umm

5:20

Few years ago I was having Cancer as an illness 😭 not untill I meant a spell caster named DR FREDRICK who helped me to be free from Cancer. If you want to be free from cancer Text DR FREDRICK on whatsapp as soon as possible (+1,617,465,5935) and testify later..

Didn't ask

@@alexandermarcus9076 Stfu

STFU

@@bigmanaaron1639 thanks. Im gaining attention. 😂

Hey so was it? Hope you’re well.

Also hoping that you're alright!

@@Mustafa-rv8cl, Thank you. The tumor was successfully removed and determined as non-cancerous. Sorry for the delayed response. I just received a notification regarding this dated post. Weird!

@@ebonyleslie7122, Thank you. The tumor was successfully removed and determined non-cancerous.

The “walnut sized” tumor was removed from my nostril about ten weeks after it was discovered. Thanks be to God, the tumor did not spread into my brain, as suggested before its removal, nor did it have a lasting effect.

***** Thats right, one more point is the sugar markers. The sugar markers within the extracellular matrix vary in cancers calls. The immune system in some people is capable of seeing these markers. There are ways to expose cancer cells.

The answer is very complex. Think of it like a pest species such as mosquitos or bacteria. If you spray for mosquitos, you will kill millions of them each time you do. In that large, heterogeneous population, there will be mosquitos that have genetic mutations that render your spray useless. Over time, the susceptible population will be wiped out and replaced by those who are immune. If you try to reuse the same spray you had used previously, it will do nothing. As you mentioned, many of our immune cells will readily identify and kill cancer cells. This represents perhaps the greatest selective challenge cancer cells face. Tumors, like the mosquitos, are a large, heterogenous population. As time goes on, cancer cells, which often have corrupted DNA repair mechanisms and an incredible rate of proliferation, begin to favor the survival of cancer cells that have mutations that prevent recognition and killing by immune cells. This process is called immunoediting. One of the primary mechanisms cancer cells are recognized is by presentation of antigens on MHC I complexes found on the surface of cells. Although mutations play a significant role, what really seems to be privileging cancer cells doesn't seem to be mutations at all; instead it is the reactivation of genes that they shouldn't be using. What governs this process is unknown. Our immune system is a powerful tool to keep us same from harm, but often it makes mistakes. Collateral damage caused by the immune system can kill us. There are many tissues, particularly in the central nervous system and the human embryo, that are vital to our species and must therefore be protected from the immune system. Many of these cells present molecules on their surface that inactivate or even kill immune cells that try to target them. Cancer is evolution incarnate; not only are tumors and cancer cells constantly acquiring mutations upon which natural selection acts, but seemingly gain a key to the protein-encoding genes perfected over the last 650 million years of vertebrate evolution.

Because those cells belongs to our body our immune system does not attack our own cells even though it becomes a cancer cell because of the genetic constitution

@@KRAkil-cs4eh not necessarily true. We have a series of markers on cells that kind of provide a "window" into the cell that show what kinds of proteins it's making. If there are viral or mutated proteins being translated, these are viewed as non self

They kill some of the cancer cells but they cann't kill all of them.

Bozeman Science please tell me if this is incorrect in your opinion? I can link you to the studies if you are interested

me too, cause actually he do explain well

Probably a bit of both. Also (this is just my guess, I hv no idea for sure), the cancer cells, if they divide enough, can restrict the function of vital organs. For example, if a cancerous mass were to occupy, say, the left lung, then, the bigger it grows, the more it restricts the function of the left lung, until, eventually, it takes up the entire lung and caused it to be completely useless, while restricting the amount of oxygen at the same time

IR will cause damage to DNA in both normal and cancer cells. Cancer cells replicate faster and if repair enzymes are unable to keep the DNA repaired and faithful cancer cells die more often due to replication errors.

natural selction

STFU

I hope it stays that way

I think cancerous cells bypass the "normal" cell checkpoints, because they're mutants. And that's why it's hard for the immune system to detect a foreign antigen, because it skips specific parts of the cycle and they just replicate uncontrollably. (I think :))

Forgot to add, that's why cell apoptosis isn't effective :) because cancerous cells are mutated

Yes, the immune system removes neoplastic cells on a daily basis. It's actually amazing once you understand all the hurdles a neoplastic cell has to get over in order to survive in situ, metastasize, and then survive in the target tissue.

cool thanks for the answer!

Ajaay Persaud Like Team Rocket said, environmental factors & health choices may lead to mutations. And these mutations will cause cancer.

uncontrolled cell growth leads to formation of a tumour and then cancer which is bad for the body

Didn't ask

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Greg Balteff what