Do You Have Internal Jugular Vein Compression? What Does that Have to Do with CCI? 

Just logged in to congratulate and thank you for this awesome video. Thanks for the knowledge, doc!

I appreciate your care in presenting it so it can be understood easily, by anyone. Thank you

Great video

Variably in symptoms depends on pressures. The heart also does this. You could do the same with a random selection in the public with calcium/blockages in their arteries and they would have no symptoms. At some point the body comes under stress and the brain is impacted. The stress on the jugular vein is too much (under certain circumstances). Then the feedback loops are impacted. Then symptoms persist. The body is trying to protect from future issues due to the pathology in the physiology. Just because it exists with no obvious symptoms doesn’t mean it’s ok. That’s like saying atherosclerosis is ok if there are no symptoms. It isn’t. And yet it is probably present in a large selection of the general public.

Thank you for making this video? Do the compression cause white matter or/and chronic small vessel ischemic disease?

Thank you for sharing! Love your videos. Does your office offer Telehealth appts. In order to get testing needed to diagnose this type of problem?

38:00 fullness, sense of fluid; 44:00 arcuate foremen

Sir please can you give your appointment so that i can consult you online

just because veins collateralize doesn't mean its healthy and not causing issues. collateralization is a sign there is an issue. if they are collateralized that means they are working harder than normal and that is not right

Hi Dr. Centeno - Can you help me understand which doctors can help me diagnose this issue? I have many of the symptoms, which have been escalating for at least a few years, but I am being dismissed or symptoms treated piece-meal by the PCPs, eye doctors, and Neurologists I have seen.

Can this compression cause pain when reading, ringing in the ears and trouble looking at screens?

8:30 Ultrasound like the doppler ultrasound ? But if the operator does it properly, it's fine, right ? Because if the patient moves like you mentioned, you just have to correct the position and the problem is gone.

Can IJV become calcified if irritated or conpressed for many years?

Can this venous compression lead to downstream complications or changes in venous flow/pressure such as weakness and numbness in the limbs and muscles below the neck? I have slight transverse ligament instability, synovial joint inflammation in the C1-C2 area, cervicla curve reversal, and likely intracranial hypertension arising from this specific problem. But whenever my C1 moves slightly abnormally to one side or too far forward I get stroke like attacks very suddenly that look like myelopathy. I don't have an upright MRI but the supine MRI did not show my dens hitting my spinal cord, or that the transverse ligament was nearly that hypermobile enough to do that according to DMX. There's no basilar invagination on my MRI. Transcranial doppler came back negative for VBI as well. So my next thought is either a CSF flow problem or a vascular one that's somehow affecting my brainstem or upper spinal cord as a result of instability.

Very good video , My right Internal jugular vein is compressed when my head is turned to the left and is ok when head turned to right ( a catheter angiogram has shown this ) , my neurological symptoms are better when keep my head turned to the right and dramatically worse when turned to the left, however my left internal jugular vein which is my dominant one is fine . Could this be the cause of my symptoms? And also is it true that the internal jugular veins collapse in the upright position and drainage is via the vertebral venous plexus. Does this mean that the internal jugular veins have no function when standing up and doing physical activity?

I am feeling very lousy worse than usual this past week if I don’t have my shoulders up in my head bent back so I can feel the drainage of the vein down my neck my heart starts to feel extremely heavy and I have to lay down please give insight

Why AO isn't a non-invasive option since C1 is usually one of the culprits of these compressions? Why jumping directly into PRP or surgery and not thinking of AO as a viable option? I'm confused on that part.

please don't forget the eagle syndrome here 

Blood flow isn’t stable. This is the issue with imaging also. Bodies move and the physiology changes. If there is pathology it is dependent on these variables. It is totally inappropriate for practitioners to think a pathology isn’t the cause of symptoms just because it exists in some without symptoms! There are genetic variabilities also.