Help For Those With Huntington's Disease - Nebraska Medicine 

elizabeth brown lets hope so brother god bless you all . It would be absolutely amazing I pray it comes true. Let’s hope these trials go well 🙏🤞🙏🤞🙏🤞

I was diagnosed with Huntington's disease in 2018 and I have tried everything I could to heal myself, but it was all to no avail, until I saw a post on a health forum this herbalist through his email and he prepared me a herbal cure for Huntington's disease and sent it to me via the delivery service of UPS from the company, when I received this herbal cure. They gave me step by step instructions on how to apply it, when I applied it according to the instructions I was totally cured of this disease within 5 months of use, I am now Huntington's disease free, all thanks to Dr. Timothy You can also contact this great herbalist for help by email: timothydouglas3612@gmail.com or contact Dr. Timothy through his whatsapp number: +1(239) 977-4390 He is also specialize in treating all kinds of diseases, VIRUS HERPES , HEPATITIS B, CANCER, BRAIN DISEASE, INFERTILITY, DIABETES EPILEPSY AND MORE ..... He will also be of help to you

I Am so sorry for you, my son is at risk because it runs in my family. They don't understand how much the family pays for charity and medical bills. I was tested at 19 and it was more than 1500 dollars

Where can i go and get a blood test for it. I know i have it an i have no insurance and i cant afford. 500 dollar test. Can you please atleast try an tell me how to do that. Surely there is someway that i can do a test so i can get health insurance

They make you pay for this test? It's free in Australia.

Hi, I do research about Huntington's disease and I came up with a hypothesis and if it is correct, then perhaps the drug decitabine could retard the onset of H.D. Now I'm trying to find articles that describe cases of patients with Myelodysplastic syndromes and Huntington's disease, to know if they were treated with Decitabine or another inhibitor of DNA Methyltransferase, e.g. 5-azacytidine. I'm sorry that my idea even if it could retard the onset of the disease, would not reverse previous existing damages in neurons. Here it is a short explanation of my hypothesys: If a cytosine in CAG triplet repeats is transformed into a thymine, due to the methylation of carbon 5' with subsequent deamination of the nitrogenous base, then one of the three possible STOP codons would be produced; the formation of stop codons within the HTT gene would explain truncated proteins in dopaminergic neurons. The more CAG repeats, the more likely cytosines could be transformed into thymine, producing TAG codons instead of CAG codons; when the relatively slow enzymes DNA glycosylase gets overwhelmed upon a threshold of about 40 CAG triplet repeats or more, stop codons would cause proteins to be truncated. Perhaps an already existing drug that inhibits the methylation of DNA, like decitabine, could retard the onset of H.D. I hope I'm right and my effort may help people. www.ncbi.nlm.nih.gov/pmc/articles/PMC3079022/

@@hannahrush3102 Hi, I do research about Huntington's disease and I came up with a hypothesis and if it is correct, then perhaps the drug decitabine could retard the onset of H.D. Now I'm trying to find articles that describe cases of patients with Myelodysplastic syndromes and Huntington's disease, to know if they were treated with Decitabine or another inhibitor of DNA Methyltransferase, e.g. 5-azacytidine. I'm sorry that my idea even if it could retard the onset of the disease, would not reverse previous existing damages in neurons. Here it is a short explanation of my hypothesys: If a cytosine in CAG triplet repeats is transformed into a thymine, due to the methylation of carbon 5' with subsequent deamination of the nitrogenous base, then one of the three possible STOP codons would be produced; the formation of stop codons within the HTT gene would explain truncated proteins in dopaminergic neurons. The more CAG repeats, the more likely cytosines could be transformed into thymine, producing TAG codons instead of CAG codons; when the relatively slow enzymes DNA glycosylase gets overwhelmed upon a threshold of about 40 CAG triplet repeats or more, stop codons would cause proteins to be truncated. Perhaps an already existing drug that inhibits the methylation of DNA, like decitabine, could retard the onset of H.D. I hope I'm right and my effort may help people. www.ncbi.nlm.nih.gov/pmc/articles/PMC3079022/

Hi. I do research about a number of diseases and I came up with a hypothesys; if it's true, then I believe that the drug Decitabine could retard the onset of Huntington disease. You may want to get this explanation to a doctor and talk about it: If a cytosine in CAG triplet repeats is transformed into a thymine, due to the methylation of carbon 5' with subsequent deamination of the nitrogenous base, then one of the three possible STOP codons would be produced; the formation of stop codons within the HTT gene would explain truncated proteins in dopaminergic neurons. The more CAG repeats, the more likely cytosines could be transformed into thymine, producing TAG codons instead of CAG codons; when the relatively slow enzymes DNA glycosylase gets overwhelmed upon a threshold of about 40 CAG triplet repeats or more, stop codons would cause proteins to be truncated. Perhaps an already existing drug that inhibits the methylation of DNA, like decitabine, could retard the onset of H.D. Glory to Jesus, for it's written that nothing was made without the Word!

are You from india

@@anupamamedia975 yes

@@swapnadevarakonda3912 from where Kerala?

@@anupamamedia975 not Kerala ....but we can travel

My family has also same disease

Who is Dr Stanley? Actually my father tested HTT positive So i m worried how to cure this ... Pls help me out ..

This is nonsense