Stanford Univerisity's Dr. Khandelwal and Family Heart Chief Medical Officer Dr. Mary McGowan outline who should have their Lp(a) tested and what to do with those results. #KnowLpa
Lp(a) is a risk factor, not an absolute sentence to cardiovascular disease. My doctor says "80% of CVD is environmental." So, as the video says, don't smoke, avoid obesity and diabetes, don't eat junk food, exercise, etc. Plenty of people with high Lp(a) values (which supposedly is inherited in a co-dominant fashion) never develop CVD nor have their parents and other close relatives.
What's the evidence for this statement: "Plenty of people with high Lp(a) values (which supposedly is inherited in a co-dominant fashion) never develop CVD nor have their parents and other close relatives."
Thanks for your presentation. As far as LP(a) levels it seem that the measured levels can differ when considering either mg/dl or nmol/L, they appear not to be interchangeable although 2.5 is being used as a converting number. Any comment on that? Thanks again
Dr. Khandelwal, are you familiar with the Lp(a) work done by interventional cardiologist, Dr. Nadir Ali, from Webster, Texas? Dr. Nadir Ali not only opines that Lp(a) has NOTHING/ZERO to do with CAUSING atherosclerotic diesase (CAD, CVD), but he describes Lp(a) as a very important molecule in the repair process of damaged tissue. Dr. Ali describes some elderly patients, with lifelong high Lp(a) levels, who he has performed coronary angiograms on, and these patients have absolutely NO plaque whatsoever.
My father,his 3 sisters and a brother all passed away av a kind of fast and powerful heart attack at their late 50s. All their children have extremely elevated LPa. Mine alon is 469 nmol/l. So, your Doctor probably has done his research on wrong people.
My LPa is 114, and I was told by BHF that in there opinion up to 300 is not considered extremely high and enough to impact on coronary health. But over 300 should be managed by lowering LDL to help stop the LPa being able to stop it travelling round the body. If its so important why isn't this test being carried out on everyone who has the cholesterol test.
There are some old studies showing that androgens (testosterone, anabolic steroid) lower lp(a) by 60-80%. People who take testosterone replacement or anabolic steroids could be benefiting from it. Has anyone studied this?
I was to be in a trial to reduce LPa (mine was in the 245 range). The followup prior to be in the trial my LPa was at 150 which was to low to be in trial. The only thing that changed was I was put on Repatha maybe 3 weeks before the followup. 150 is still high but it's been in that 240..250 range for about 10 years.
Best treatment for atherosclerosis: Vitamin C Why do only coronary arteries clog with cholesterol and not veins or small capillaries? Scurvy = No vitamin C in your diet. Symptom: Your blood vessels break and you bleed to death. Think of the sailor of the past. Scurvy of the heart = Just enough vitamin C from food. Symptom: Arteries around the heart are not strong enough to resist high blood pressure. Damage is repaired with cholesterol LP(a) to prevent worse. After years of repair, your arteries become clogged. Enough vitamin C supplement of at least 3000 mg. per day gives strong and flexible arteries. Cholesterol is not needed as a repair agent. Cholesterol in your coronary arteries is broken down and burned in your liver. Source RU-vid: Cardiovascular disease and vitamin C (Dr. Rath Foundation) or Breakthrough Towards The Natural Control Of Cardiovascular Disease - Dr. Rath's 2018 Cyprus Lecture
