Statement/observation around 27;20 is worth repeating... "Carb determines small LDL while saturated fat determines large LDL. The advice to avoid saturated fat effectively set up type B phenotype in the population over the last 50 years.
More accurately, "high sugar determines..." Study subjects on the "high carb diet" ate 75% of calories from carbohydrates, half of which were simple sugars.
You didn’t define your terms. Exactly what do you mean by “low” carbohydrate? Meat given as a percentage but what about in grams or ounces? In what context were the experiments performed? If a dietary daily intake in based on an appropriate number of balanced calories, such that the body is not overwhelmed with calories, do these conclusions still hold? What about an experiment that focuses on monounsaturated fat being higher than saturated fat? Etc. what about LpA?
May be an LDL-receptor issue (i.e. ApoB containing Lipoprotein particles don't get removed from the blood stream by the liver). I don't know if that matters though...
I would rather ask: would it bring my risk down? My answer would be: there seems to be a strong link (apparently through a number of individual mechanisms) between chronically elevated insulin and atherosclerosis / CHD.
