Man, you are saving lives. I don't know where would I've been if I haven't found your leactures! Thank you so much for all your work, please keep on rocking!!!
Jama article 2021 reveals that more pills for so-called high BP results in more morbidity and worse outcomes. NonRenal, essential Hypertension is a fake disease invented for big pharma to
Thanks for this presentation. As a CHF patient this helped me understand why I am on 3 different diuretic medicines, who they work and explained side effects much better then someone just telling me this will make you urinate more but get rid of the access fluid. I appreciate you posting this.
@@arsalanazhar3340 1 year, I am alive but doctors are wanting me on hospice now. I am fighting it, trying to do my best but I know how it ends. Then I'm ready to see my heavenly father.
Whenever I need a lecture on pharmacology I always rush to your channel This is awesome, I wish you had lectures on malaria and other pharmacological topics Cheers 🥂
Thank you sir, this helped me a lot:) But I request you to make videos about the anti-microbials and CNS drugs as they are huge topics and difficult to cover quickly, I have my finals soon 😭
Diuretics do not reduce edema in legs. Where does the urine output come from? The blood? Is that why blood thinners are prescribed? Even when low blood pressure is a fact?
Tq very much sir... But i have a doubt... At 12:21 (regarding cause of hyper calcemia) increase in sodium reabsorption at PCT must decrease calcium reabsorption....as both are positively charged ions...??
Honestly you are the best one I have ever heard in Diuretics so now I can suggest you to my friends to understand this lecture ❤❤ your brother from Egypt🇪🇬
You are a very good teacher. Good explanation, great summary, this is really speed pharmacology. I love the fact that you always start with physiological it helps to set the pace
Did I forget to thank you for helping me get through my exams without palpitations and also understand the concepts better?Yes,I did. Thank you very very much.Please keep doing an amazing job. P.S:Hearing your voice a month after my exams kinda hit me hard.Like it felt so familiar.
I believe blocking the NKCCl2 cotransporter creates a less positive lumen potential, leading to magnesium and the other cations being retained in the lumen, and excreted. It wouldn't make sense that if the lumen potential is more positive that those cations would be retained, right?
Sir, i have a doubt, in the book, the mechanism of action of amiloride and triamterene is given as inhibitors of renal epithelial sodium channel but you have explained it as competitive antagonist of aldosterone, please explain if it is the same or not??? Thank you
Well u said spironolactone will stimulate receptos for testosterone n progestrone.. But its not right.. Spironolactone resembles testosterone in structure n hence can competitively inhibit testosterone n progestrone receptors which will lead to impotence n gynecomastia in males n menstrual irregularities in females n moreover bcz it inhibits testosterone receptprs thats why it is also used for tx of hirsuitism
Thanks so much for your videos. They are amazing. Doesn't water tend to move from a higher osmolarity (more concentrated) to a lower osmolarity (more dilute) in osmosis rather than a lower osmolarity to a higher osmolarity as stated in the video?
