MedCat is focused on providing you free MCAT prep videos. I scored in the 99th percentile, qualifying me to teach the MCAT at any major test prep company, and I have many hours of live tutoring experience specifically focusing on the MCAT. My methodology: analyze what the AAMC thinks is important based on their practice materials and explain the most high yield content to you. _____________________________ All content and media on this channel is created and published online for informational purposes only. It is not intended to be a substitute for professional medical advice and should not be relied on as health or personal advice.
Thank you i have just one question, why aren't the yellow dominant alleles ignored in the probability. I'm assuming its because the problem stated that 2 yellow flies are going to be mated?
How's it going everyone! I'm making some shorter videos with the thought that seeing this video once will yield you hopefully at least one question on the MCAT. Let me know how you feel about the videos with shorter format.
i usually dont comment on youtube videos but im taking my mcat the day after tomorrow and you rly made this click for me. ive been binging your videos since i wanted to do some light content review and ive learned so much. thank you and i hope youre doing well in med school
Interesting. Actually. Very interesting. And also. Quite useful. Thanks. As here. I have probably. Accidentally poisoned myself. Yet. From memory. I thought of a great remedy and through. Deduction. Perhaps can think of the cause of this discomfort I feel. Now. Unfortunately. I am slightly disabled and canget out to the store like I used to. Where I was checking out the packets of foods as. I was buying them. So. I ordered home delivery actually and by coincidence. I happen to know a bit about the culture of Coca-Cola. And through research online. I discovered some background to a drink named Dr. Pepper. Now . I am lying here . Feeling a little discomfort. Behind my breast bone. A classic sympton of Phosphoric Acid poisoning. According to online reports. My teeth also feel a little tender perhaps due to. As usual. I forgot to brush them for a while. Which is not usually much of a problem. So. Remembering what I read of Dr Pepper. It was formulated in Texas by a Pharmacist possibly who sold sodas. 2. It sold well in Mississippi near to a point where it was manufactured . Coca Cola . Is partly Berkshire Hathaway owned and Coke is marketed through parties in the stadiums of America and through The Coca Cola lorry near Christmas. Here in Plymouth. When a lorry full of Coke is driven up. Onto Plymouth Hoe. Plymouth. Devon. England. United Kingdom. British Isles. Off Continental Western Europe. So. The sodium content of the formulations. I am guessing is crucial. However it's likely that. Coca Cola distribution. Just distributes a syrup for adding to local water supplies. From memory sodium content of water varies. As does Chlorine conyent. I guess. High Sodium content and a lower chlorine content might be suitable here so. From memory . I am guessing that fits the profile of Evian. Mineral water. Thus. I should seek Evian as a mixer for Tge Cratr of Dr. Pepper that I bought myself as a treat. Yet. That's a bit of a drag to walk to the store and formulate my own improved version of Dr. Pepper right ?
Why is concentration of glucose in the lumen lower than that of the intestinal epithelial cell? After a carb diet, won’t the luminal glucose be higher? I always get confused with this.
**disclaimer: this process is probably way more complicated than this, and there could be wrong suppositions I make, but this is how I think of it in terms of MCAT concepts. I would disagree with the statement that after a carb-rich meal that the blood luminal glucose would be higher than that of the inside of the intestinal cell. The blood luminal glucose is "high" (relative to fasting glucose, but not to the inside of the intestinal cell) after a carbohydrate-rich meal because of the intestinal epithelial cells having a higher intracellular concentration of glucose! For passive transport to occur, the intracellular concentration must be higher than the blood glucose concentration. You could also think of it in terms of ins and outs. The way the system is set up, more glucose is coming in the apical side of the cell than is leaving via the basal side of the cell, so the enterocyte (should) have more glucose than the blood.
@@medcatmcat Thank you so much for this detailed explanation. I do understand that the concentration of glucose in the intestinal epithelial cell is higher than that in the blood (basal side) for facilitated diffusion to occur down the concentration gradient. However, what I don’t understand is that, after a high carb meal, wouldn’t the concentration of glucose in the INTESTINAL LUMEN be greater than that in the intestinal cell (on the apical side)? Why do we need secondary active transport for glucose here and why is glucose going UP its concentration gradient?
Around 6:21, you say gotten rid of a negative charge when the Lysine residue was acetylated, but it was a positive charge (basic) that was removed, thus lowering the pI because it became less basic or more acidic
On semeiotics book I read: "Use the bell to listen when measuring patient's pressure" and I was like... WTF is a Bell? I HAVE NO BELL... Then I found this video... Thank you!
do you have channel recommendations to study this part for the mcat? so that I can have a comprehensive grasp on the material that is most likely to be tested?
As far as RU-vid, I'm not quite sure to be totally honest. I think the best way to really get a comprehensive understanding would be to use a text like Kaplan or Princeton Review and supplement with RU-vid search as needed--I would not feel comfortable with any channel that claims it is truly "comprehensive."
@@medcatmcat I understand that its discouraging to put effort and not get a lot of views, but thats just the nature of making educational content on youtube nowadays. Ive sent this to my friends so my whole community will be benefitting from your work. The way i see it…youre a hidden gem for people who want to do good. Theres some merit in that too!
I still am quite confused as to why histidine (pKa = 6) isn't considered weakly acidic but basic? and you clearly indicated that arginine and lysine having overall charge of +1 from the diagram 8:20 but you call them polar (not charged)? Great video :) I see you uploaded them 3 yrs ago but I still find it very useful thx
Hello! I'm glad you find them useful. The classification of histidine is frustrating. It is a good reminder of another high-yield concept, though, which is the definition of pKa: the pH at which an equal amount of protonated and nonprotonated species will exist of that particular molecule. If we solve the Henderson-Hasselbach equation and give [A-] and [HA] the same value, we see that pKa must equal pH. This reminds us of an even more fundamental concept-not easy, but fundamental-which is that when we talk about histidine, singular, we actually mean an unfathomable number of histidine molecules. For simplicity, we say histidine is neutral at physiologic pH, 7.4, because the pH > pKa, and the proton that histidine sometimes likes to have is taken by the solution. However, because there are a ton of histidine molecules, some of them are going to be protonated, and in fact, because the pKa is reasonably close to the pH, a nonsignificant number of them will be found protonated in the body. What's more is that the entirety of the body is not homogenous, and some local enzymes and environments in the body will favor one form over another-a little bit of hand-waving here since this is above my pay grade. Let me know if that makes sense.
If the 'constant bell chime' (first stimulus) is replaced entirely by a sharp ringing would it still be dishabituation? Wouldnt the chime have to continue simultaneously with the ringing for dishabituation to occur?
It would not be, as the stimulus that had been habituated, or "gotten used to," would just disappear, so you wouldn't be able to notice it again. You must perceive a stimulus similarly to how you did before you were habituated, or before you had gotten used to something.
Further, if you are worried about this for the MCAT, I seriously doubt that they would present anything other than a classical situation of dishabituation, so I wouldn't perseverate on it for too long, as there is a lot more to learn-unless you are genuinely interested, in which case, hats off to your love of learning!
It is worth noting that for that model, the diaphragms are tunable. So you will get bell and diaphragm sounds depending on how much pressure. So technically it is a bell, even if you have the peds diaphragm attached.
You can do that for sure, but depending on patient sensitivity and your preference, I wanted to show the "proper" bell. To my ear, using the bell sounds totally different than pressing hard with the diaphragm.
I have not used the Harvey stethoscopes more than a handful of times, but I did not note a huge difference. I'd say if you like a bit more of a steampunk look to your stethoscope, the Harvey is your move. I also am not sure if the Harvey has a non-chill bell sleeve, so it might be cold for patients without that (though I could be wrong, I haven't seen one for that model).
